Previous works stated that only two membranes were Lapatinib present, the vacuolar membrane and one of the two bacterial membranes. The absence of the cell wall was related to the special vertical transmission of the endosymbionts in whiteflies. In this work,
we present electron microscopic studies showing a complete cell envelope in ‘Ca. Portiera aleyrodidarum’ from the whitefly Bemisia tabaci. Additionally, comparison of the inferred metabolism from the gene content did not show any difference in cell envelope biogenesis compared with the closely related three-membrane endosymbionts ‘Candidatus Carsonella ruddii’ and ‘Candidatus Evansia muelleri’ Xc1. Our results rule out the proposal that ‘Ca. Portiera aleyrodidarum’ is an exception to the three-membrane
system. “
“Kosakonia radicincitans (formerly known as Enterobacter radicincitans), an endophytic bacterium was isolated from the symptomatic tissues of bacterial wilt diseased banana (Musa spp.) plant in Malaysia. The total genome size of K. radicincitans UMEnt01/12 is 5 783 769 bp with 5463 coding sequences (CDS), 75 tRNAs, and 9 rRNAs. The annotated draft genome of the K. radicincitans UMEnt01/12 strain might shed light on its role as a bacterial wilt-associated bacterium. “
” Gerd Döring, Professor of Medical Microbiology and Hygiene at the Selumetinib solubility dmso University of Tübingen (Germany), was very much looking forward to attending the 14th International Conference on Pseudomonas, to which he had been invited and where he was going to chair a session on cystic
fibrosis (CF) and lead a discussion on antibiotic therapy against Pseudomonas aeruginosa infections, in September 2013. But fate had it otherwise. Gerd died on 2 July 2013, after a malignant melanoma had spread to his lung with uncanny speed. Gerd Döring was born in Nürnberg on 30 crotamiton August 1948, and studied chemistry at the University of Tübingen, where he obtained a PhD for his work on transition metal complexes in 1978. From 1977 to his death, Gerd mostly worked at the Hygiene Institute in Tübingen, only interrupted by scientific visits to Niels Høiby’s laboratory in Copenhagen in the 1980s and 1990s and by a study leave in Lyon in 1992. Under the guidance of the former director of the Hygiene Institute, Konrad Botzenhart, Gerd Döring developed a keen interest in P. aeruginosa and in the chronic infections that this bacterium causes in the lung of CF patients. His post doctoral ‘habilitation’ thesis published in 1986 dealt with pathogenic mechanisms of P. aeruginosa (in particular, proteases), their regulation, and consequences for inflammation. In the same year, one of us (DH) met Gerd for the first time at a symposium that he organized on ‘Basic research and clinical aspects of P. aeruginosa’ in Tübingen. At that time, Gerd was intrigued by observations indicating that P. aeruginosa must be well adapted to hypoxic conditions, in particular in the CF lung, and so we decided to test whether the ability of P.